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Tp53 deletion in B lineage cells predisposes mice to lymphomas with oncogenic translocations

MAW Rowh, A DeMicco, JE Horowitz, B Yin… - Oncogene, 2011 - nature.com
Inactivating Tp53 mutations are frequent genetic lesions in human tumors that harbor genomic
instability, including B lineage lymphomas with IG translocations. Antigen receptor genes are
assembled and modified in developing lymphocytes by RAG/AID-initiated genomic ...
All 3 versions

Function of leukemogenic mixed lineage leukemia 1 (MLL) fusion proteins through distinct partner protein complexes

D Biswas, TA Milne, V Basrur… - Proceedings of the …, 2011 - National Acad Sciences
A number of acute leukemias arise from fusion of the mixed lineage leukemia 1 protein
(MLL) N terminus to a variety of fusion partners that have been reported to reside in one or more
poorly defined complexes linked to transcription elongation through in- teractions with the ...

Lineage-specific Effects of 1, 25-Dihydroxyvitamin D3 on the Development of Effector CD4 T Cells

MT Palmer, YK Lee, CL Maynard, JR Oliver… - Journal of Biological …, 2011 - ASBMB
Vitamin D deficiency is implicated in autoimmune disease. We therefore evaluated the effects
of 1α,25-dihydroxyvitamin D 3 (1,25-D 3 ), the active form of vitamin D, on the development of
T helper 1 (Th1), Th17, and Th9 cells, which are implicated in the pathogenesis of ...
Cited by 3 - Related articles - All 4 versions

Transcription factor-induced lineage selection of stem-cell-derived neural progenitor cells.

L Panman, E Andersson, Z Alekseenko… - Cell stem …, 2011 - ncbi.nlm.nih.gov
The generation of specific types of neurons from stem cells offers important opportunities in regenerative
medicine. However, future applications and proper verification of cell identities will require stringent
ways to generate homogeneous neuronal cultures. Here we show that transcription ...
Cited by 1

The gata1/pu. 1 lineage fate paradigm varies between blood populations and is modulated by tif1γ

R Monteiro, C Pouget… - The EMBO Journal, 2011 - nature.com
Lineage fate decisions underpin much of development as well as tissue homeostasis in the
adult. A mechanistic paradigm for such decisions is the erythroid versus myeloid fate decision
controlled by cross-antagonism between gata1 and pu.1 transcription factors. In this ...
Cited by 1 - Related articles - All 3 versions

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