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[HTML] Classification of pediatric pulmonary hypertensive vascular disease: Does it need to be different from the adult classification?

[HTML] from pulmonarycirculation.orgRJ Barst - Pulmonary Circulation, 2011 - pulmonarycirculation.org
Twenty-five years later, a second World Symposium on Pulmonary Arterial Hypertension
(PAH) was held in 1998 in Evian, France. Based on the research that had ensued in studying
the pulmonary circulation since 1973, by 1998 significant advances had been made in ...

[HTML] Computational models of the pulmonary circulation: Insights and the move towards clinically directed studies

[HTML] from pulmonarycirculation.orgMH Tawhai, AR Clark… - Pulmonary …, 2011 - pulmonarycirculation.org
Biophysically-based computational models provide a tool for integrating and explaining experimental
data, observations, and hypotheses. Computational models of the pulmonary circulation have
evolved from minimal and efficient constructs that have been used to study individual ...

[HTML] Pulmonary Circulation: A new venue for communicating your findings, ideas and perspectives

[HTML] from pulmonarycirculation.orgJY JX, NW Morrell… - Pulmonary …, 2011 - pulmonarycirculation.org
The pulmonary circulation, once referred to as the lesser circulation, is now a circulatory system
that medical and research professionals (as well as patients) can no longer ignore. Based on
the World Health Organization's (WHO) annual report in 2010, pulmonary vascular ...

Genetic evidence for NAD (P) H: quinone oxidoreductase 1-catalyzed quinone reduction on passage through the mouse pulmonary circulation

BJ Lindemer, RD Bongard… - American Journal of …, 2011 - Am Physiological Soc
The quinones duroquinone (DQ) and coenzyme Q 1 (CoQ 1 ) and quinone reductase inhibitors
have been used to identify reductases involved in quinone reduction on passage through the
pulmonary circulation. In perfused rat lung, NAD(P)H:quinone oxidoreductase 1 (NQO1) ...
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[HTML] … of mutant BMPR-II in pulmonary endothelial cells promotes apoptosis and a release of factors that stimulate proliferation of pulmonary arterial smooth muscle …

[HTML] from pulmonarycirculation.orgX Yang, L Long, PN Reynolds… - Pulmonary …, 2011 - pulmonarycirculation.org
Mutations in the bone morphogenetic protein type II receptor gene (BMPR-II) are the major cause
of heritable pulmonary arterial hypertension (PAH). Although both endothelial and smooth muscle
cell BMPR-II dysfunction have been seen to contribute to pulmonary hypertension in vivo, ...
Cited by 1 - Related articles

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