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A new phenotypic manifestation of familial adenomatous polyposis

DL Edelstein, FM Giardiello, A Basiri, LM Hylind… - Familial cancer, 2011 - Springer
... In addition, evaluation by immunohistochemistry of intestinal polyps in mice heterozygous for
the multiple intestinal neoplasia gene (Min/?) (the murine model of human familial adenomatous
polyposis) revealed an increase and redistribution of VEGF-A in the proximity of those ...
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Investigation of the Birt-Hogg-Dubé tumour suppressor gene (FLCN) in familial and sporadic colorectal cancer

[PDF] from peerproject.euE Maher, M Nahorski, D Lim, L Martin, J Gille… - 2011 - repository.peerproject.eu
... Gastrointestinal polyposis with esophageal polyposis is useful for early diagnosis of Cowden's ...
Inhibition of the mTORC1 pathway suppresses intestinal polyp formation and reduces mortality ...
Implications of familial colorectal cancer risk profiles and microsatellite instability status ...
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Modified Multivisceral Transplantation With Spleen-Preserving Pancreaticoduodenectomy for Patients With Familial Adenomatous Polyposis “Gardner's Syndrome”

RJ Cruz Jr, G Costa, GJ Bond, K Soltys… - …, 2011 - journals.lww.com
... of visceral transplantation has added a new therapeutic dimension to the conventional algorithmic
management of patients with familial adenomatous polyposis (FAP) ( 1, 2 ). Different modalities
of the procedure are often required to rescue patients with intestinal failure and ...
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Aspirin and Familial Adenomatous Polyposis: Coming Full Circle

AT Chan - Cancer Prevention Research, 2011 - AACR
... 2 overexpression and tumor formation are blocked by sulindac in a murine model of familial
adenomatous polyposis. ... 56:710–4. Abstract/FREE Full Text. 16.↵: Barnes CJ,; Lee M. .
Chemoprevention of spontaneous intestinal adenomas in the adenomatous polyposis coli Min ...
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Stem cell markers characterise familial adenomatous polyposis

T Wang, KG Yeoh… - Gut, 2011 - gut.bmj.com
... receptor 5 (Lgr5), Musashi-1, Bmi1 and CD44, and the severity of intestinal polyposis in Apc 1322T
mice, which had a submaximal level of active/nuclear β-catenin. Their findings deserve to be
validated in clinical specimens. Patients with familial adenomatous polyposis (FAP ...

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