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Exercise and PGC-1 [alpha]-Independent Synchronization of Type I Muscle Metabolism and Vasculature by ERR [gamma]

VA Narkar, W Fan, M Downes, RT Yu, JW Jonker… - Cell Metabolism, 2011 - Elsevier
How type I skeletal muscle inherently maintains high oxidative and vascular capacity in the absence
of exercise is unclear. We show that nuclear receptor ERRγ is highly expressed in type I muscle
and, when transgenically expressed in anaerobic type II muscles (ERRGO mice), dually ...
Cited by 2 - Related articles - All 2 versions

Regulation of cancer cell metabolism

RA Cairns, IS Harris… - Nature Reviews Cancer, 2011 - nature.com
Interest in the topic of tumour metabolism has waxed and waned over the past century of cancer
research. The early observations of Warburg and his contemporaries established that there are
fundamental differences in the central metabolic pathways operating in malignant tissue. ...
Cited by 20 - Related articles - All 3 versions

The Logic Linking Protein Acetylation and Metabolism

L Guarente - Cell Metabolism, 2011 - Elsevier
It is now apparent that a large number of cellular proteins are acetylated. For example, recent
studies have uncovered acetylation of a high fraction of mitochondrial proteins (Kim et al.,
2006), as well as most metabolic enzymes in the cytosolic/nuclear pool ([Wang et al., ...

Wnt signaling regulates hepatic metabolism.

H Liu, MM Fergusson, JJ Wu, II Rovira… - Science …, 2011 - ncbi.nlm.nih.gov
The contribution of the Wnt pathway has been extensively characterized in embryogenesis,
differentiation, and stem cell biology but not in mammalian metabolism. Here, using in vivo
gain- and loss-of-function models, we demonstrate an important role for Wnt signaling in ...
Cited by 1 - Related articles

FGF15/19 Regulates Hepatic Glucose Metabolism by Inhibiting the CREB-PGC-1 [alpha] Pathway

MJ Potthoff, J Boney-Montoya, M Choi, T He… - Cell Metabolism, 2011 - Elsevier
Regulation of hepatic carbohydrate homeostasis is crucial for maintaining energy balance in
the face of fluctuating nutrient availability. Here, we show that the hormone fibroblast growth
factor 15/19 (FGF15/19), which is released postprandially from the small intestine, inhibits ...
Cited by 1 - All 2 versions

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