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[HTML] Inhibition of lipolysis in Type 2 diabetes normalizes glucose disposal without change in muscle glycogen synthesis rates

[HTML] from clinsci.orgE Lim, K Hollingsworth, F Smith, P Thelwall… - Clinical …, 2011 - submit.clinsci.org
Suppression of lipolysis by acipimox is known to improve insulin-stimulated glucose
disposal, and this is an important phenomenon. The mechanism has been assumed to be an
enhancement of glucose storage as glycogen, but no direct measurement has tested this ...
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Syringe disposal among injection drug users in San Francisco

[HTML] from aphapublications.orgLD Wenger, AN Martinez… - … journal of public …, 2011 - ajph.aphapublications.org
Needlestick injuries resulting from injection drug users (IDUs) improperly disposing of syringes
present a potential risk of transmission of viral infections such as hepatitis and HIV to community
members, sanitation workers, law enforcement officers, and hospital workers. 1–8 There ...
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Nonoxidative Free Fatty Acid Disposal Is Greater in Young Women than Men

C Koutsari, R Basu, RA Rizza, KS Nair… - Journal of Clinical …, 2011 - Endocrine Soc
Context:Large increases in systemic free fatty acid (FFA) availability in the absence of a corresponding
increase in fatty acid oxidation can create a host of metabolic abnormalities. These adverse responses
are thought to be the result of fatty acids being shunted into hepatic very low-density ...
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LLNL Input to SNL L2 MS: Report on the Basis for Selection of Disposal Options

M Sutton, JA Blink… - 2011 - osti.gov
This document was prepared as an account of work sponsored by an agency of the United States
government. Neither the United States government nor Lawrence Livermore National
Security, LLC, nor any of their employees makes any warranty, expressed or implied, or ...
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Alpha-1 Antitrypsin Deficiency: Importance of Proteasomal and Autophagic Degradative Pathways in Disposal of Liver Disease-Associated Protein Aggregates

D Perlmutter - Annual Review of Medicine, 2011 - annualreviews.org
Alpha-1-antitrypsin (AT) deficiency is the most common genetic cause of liver disease in
children. The primary pathological issue is a point mutation that renders an abundant hepatic
secretory glycoprotein prone to altered folding and a tendency to polymerize and ...
Cited by 1 - Related articles - All 2 versions

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