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A decisive function of TGF {beta}/Smad signaling in tissue morphogenesis and differentiation of human HaCaT keratinocytes

S Buschke, HJ Stark, A Cerezo… - Molecular Biology of …, 2011 - Am Soc Cell Biol
The mechanism by which TGFβ regulates differentiation in human epidermal keratinocytes is
still poorly understood. To assess the role of Smad signaling, we engineered human HaCaT
keratinocytes either expressing siRNA against Smads2, 3, 4 or overexpressing Smad7 ...
All 2 versions

A decisive function of transforming growth factor-{beta}/Smad signaling in tissue morphogenesis and differentiation of human HaCaT keratinocytes

[HTML] from molbiolcell.orgS Buschke, HJ Stark, A Cerezo… - Molecular Biology of …, 2011 - Am Soc Cell Biol
The mechanism by which transforming growth factor-β (TGFβ) regulates differentiation in human
epidermal keratinocytes is still poorly understood. To assess the role of Smad signaling, we engineered
human HaCaT keratinocytes either expressing small interfering RNA against Smads2, 3, ...
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[HTML] Fat in the skin: Triacylglycerol metabolism in keratinocytes and its role in the development of neutral lipid storage disease

[HTML] from nih.govFPW Radner, S Grond, G Haemmerle… - Dermato- …, 2011 - ncbi.nlm.nih.gov
Keratinocyte differentiation is essential for skin development and the formation of the skin permeability
barrier. This process involves an orchestrated remodeling of lipids. The cleavage of precursor
lipids from lamellar bodies by β-glucocerebrosidase, sphingomyelinase, phospholipases ...
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[HTML] Chitin Modulates Innate Immune Responses of Keratinocytes

[HTML] from plos.orgB Koller, AS Müller-Wiefel, R Rupec, HC Korting… - PloS one, 2011 - dx.plos.org
... J Invest Dermatol 121: 1389–96. Find this article online; Baker BS, Ovigne JM, Powles AV,
Corcoran S, Fry L (2003) Normal keratinocytes express Toll-like receptors (TLRs) 1, 2 and 5:
modulation of TLR expression in chronic plaque psoriasis. Br J Dermatol 148: 670–679. ...
Cited by 1 - Related articles - Cached - All 4 versions

STAT3-dependent effects of IL-22 in human keratinocytes are counterregulated by sirtuin 1 through a direct inhibition of STAT3 acetylation

R Sestito, S Madonna, C Scarponi, F Cianfarani… - The FASEB Journal, 2011 - FASEB
IL-22 has a pathogenetic role in psoriasis, where it is responsible for the altered proliferation
and differentiation of keratinocytes and induces inflammatory molecules. The IL-22-induced effects
are mediated by STAT3, whose activity is proportional to acetylation in lysine (Lys)685 ...
Related articles - All 3 versions

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