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Methods for decreasing osteoclast formation or bone resorption using an antibody to osteoprotegerin binding protein

WJ Boyle - US Patent 7,923,008, 2011 - Google Patents
IIIIIIH US007923008B2 (12) United States Patent Boyle (io) Patent No.: (45) Date of Patent: US
7,923,008 B2 *Apr. 12, 2011 (54) METHODS FOR DECREASING OSTEOCLAST FORMATION
OR BONE RESORPTION USING AN ANTIBODY TO OSTEOPROTEGERIN BINDING ...
Related articles - All 2 versions

What Makes an Osteoclast Special?

G Karsenty - Developmental Cell, 2011 - Elsevier
A paper published in Developmental Cell that I have continuously enjoyed over the years is
“Induction and Activation of the Transcription Factor NFATc1 Integrate RANKL Signaling in Terminal
Differentiation of Osteoclasts” by Takayanagi et al. I like this paper for many reasons. The ...
All 2 versions

AGENTS FOR INHIBITING OSTEOCLASTOGENESIS AND/OR OSTEOCLAST ACTIVATION

A Karadimitris, N Horwood… - US Patent …, 2011 - freepatentsonline.com
Agents, which are both ceramide glucosyltransferase inhibitors and glucosidase inhibitors, may
inhibit osteoclastogenesis and/or reduce osteoclast activation and, therefore, may be useful for
osteolytic activity and bone loss in subjects with conditions, such as multiple myeloma.
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Ovariectomy disregulates osteoblast and osteoclast formation through the T-cell receptor CD40 ligand

[HTML] from pnas.orgJY Li, H Tawfeek, B Bedi, X Yang… - Proceedings of the …, 2011 - National Acad Sciences
The bone loss induced by ovariectomy (ovx) has been linked to increased production of osteoclastogenic
cytokines by bone marrow cells, including T cells and stromal cells (SCs). It is presently unknown
whether regulatory interactions between these lineages contribute to the effects of ovx in ...
Cited by 1 - Related articles - All 4 versions

Kindlin-3–mediated signaling from multiple integrin classes is required for osteoclast-mediated bone resorption

S Schmidt, I Nakchbandi, R Ruppert… - The Journal of Cell …, 2011 - jcb.rupress.org
The blood cell–specific kindlin-3 protein is required to activate leukocyte and platelet
integrins. In line with this function, mutations in the KINDLIN-3 gene in man cause immunodeficiency
and severe bleeding. Some patients also suffer from osteopetrosis, but the underlying ...
Cited by 2 - Related articles - All 4 versions

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